Draft Genome Sequence of a Stable Mucoid Strain of Pseudomonas aeruginosa PAO581 with a mucA25 Mutation
Journal Article
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· Genome Announcements
- Marshall Univ., Huntington, WV (United States). Joan C. Edwards School of Medicine. Depts. of Biochemistry and Microbiology
- Univ. of Edinburgh, Scotland (United Kingdom). Cystic Fibrosis Lab.
- Los Alamos National Lab. (LANL), Los Alamos, NM (United States). Genome Science Group
- Marshall Univ., Huntington, WV (United States). Joan C. Edwards School of Medicine. Depts. of Biochemistry and Microbiology; Marshall Univ., Huntington, WV (United States). Joan C. Edwards School of Medicine. Dept. of Pediatrics; Progenesis Technologies, LLC, Huntington, WV (United States).
A mutation in the mucA gene, which encodes a negative regulator of alginate production in Pseudomonas aeruginosa, is the main mechanism underlying the conversion to mucoidy in clinical isolates from patients with cystic fibrosis (CF). Here, we announce the draft genome sequence of the stable alginate-overproducing mucoid strain P. aeruginosa PAO581 with a mucA25 mutation, a derivative from the nonmucoid strains P. aeruginosa PAO381 and PAO1.
- Research Organization:
- Los Alamos National Lab. (LANL), Los Alamos, NM (United States)
- Sponsoring Organization:
- USDOE Office of Science (SC), Biological and Environmental Research (BER). Biological Systems Science Division; National Institutes of Health (NIH)
- Grant/Contract Number:
- P20RR016477; P20GM103434
- OSTI ID:
- 1629281
- Journal Information:
- Genome Announcements, Vol. 1, Issue 5; ISSN 2169-8287
- Publisher:
- American Society for MicrobiologyCopyright Statement
- Country of Publication:
- United States
- Language:
- English
Genome sequence of Pseudomonas aeruginosa PAO1161, a PAO1 derivative with the ICEPae1161 integrative and conjugative element
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