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Effect of influenza infection on the phagocytic and bactericidal activities of pulmonary macrophages

Journal Article · · Infect. Immun.; (United States)
OSTI ID:6566464

The effect of mouse-adapted influenza A/PR/8/34 virus on pulmonary macrophage function was evaluated by using an in vitro system which allowed direct virus interaction with macrophages and then separate analysis of the steps required for bacterial clearance by macrophages. Infection of macrophages with this virus resulted in the appearance of a hemagglutinating activity on the macrophage surface; expression of this activity was inhibited by amantadine, 2-deoxyglucose, and cycloheximide and by pretreatment of the virus inoculum with with ultraviolet light and specific antiserum. After influenza infection, net ingestion of viable Staphylococcus aureus by macrophage monolayers was unaltered and there was no change in the fraction of the monolayer which ingested cocci over a wide range of bacterial inputs. Influenza-infected microphages also inactivated intracellular S. aureus at a rate indistinguishable from controls. Therefore, these in vitro studies do not support the hypothesis that the defect in pulmonary antibacterial mechanisms associated with influenza infections results from a direct effect of virus infection on either the phagocytic or bactericidal activity of resistant pulmonary macarophages.

Research Organization:
Univ. of Iowa Hospitals, Iowa City
OSTI ID:
6566464
Journal Information:
Infect. Immun.; (United States), Journal Name: Infect. Immun.; (United States) Vol. 26:2; ISSN INFIB
Country of Publication:
United States
Language:
English