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Decreased labeling of amino acids by inhibition of the utilization of (/sup 3/H, /sup 14/C)glucose via the hexosemonophosphate shunt in rat brain in vivo

Journal Article · · Neurochem. Res.; (United States)
DOI:https://doi.org/10.1007/BF00963984· OSTI ID:6132596

Treatment of rats with 6-aminonicotinamide showed a small but significant decrease in the labeling of amino acids in the brain after injection of (/sup 3/H)acetate. The results of these experiments also gave evidence of the presence of (/sup 3/H)glucose and (/sup 3/H)acetate, and an increase in (/sup 3/H)glucose content in the brain of 6-aminonicotinamide treated rats. To apportion the contribution of (/sup 3/H)glucose formed by gluconeogenesis from (/sup 3/H)acetate to the labeling of amino acids a method was formulated based on the measurement of radioactivity of amino acids, lactate and free sugars in brain after injection of (6-/sup 3/H)glucose or (1-/sup 3/H)glucose relative to that after co-injection of (U-/sup 14/C)glucose or (2-/sup 14/C)glucose. These findings indicated that a significant decrease in the labeling of amino acids from (U-14C)glucose reported previously and again confirmed using (1-/sup 3/H), (6-/sup 3/H), (2-/sup 14/C) or (U-/sup 14/C)glucose in the present investigation was not due to the inhibition of the activities of enzymes of the citric acid cycle. These results support the postulated role of the hexosemonophosphate shunt for the utilization of glucose in providing neurotransmitter amino acids glutamate and gamma-aminobutyrate.

Research Organization:
St. George's Hospital Medical School, London, England
OSTI ID:
6132596
Journal Information:
Neurochem. Res.; (United States), Journal Name: Neurochem. Res.; (United States) Vol. 9:3; ISSN NERED
Country of Publication:
United States
Language:
English