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Vascular permeability and late radiation fibrosis in mouse lung

Conference · · Radiat. Res.; (United States)
OSTI ID:5227733

It has been suggested that fibrosis which developed after irradiation is caused by increases in vascular permeability. Plasma proteins leak into irradiated tissue where fibrinogen may be converted into fibrin which is gradually replaced by fibrous tissue. Vascular and fibrotic changes in mouse lung were investigated after X irradiation of the right hemithorax. Blood volume and accumulation of extravascular proteins were measured using indium (/sup 111/In)-labeled red cells, iodinated (/sup 131/I) albumin, and iodinated (/sup 125/I) fibrinogen. During the first few months after X rays, lung blood volume decreased to a plateau which depended on radiation dose (10-25 Gy). Small increases in extravascular albumin and fibrinogen occurred at 1-12 weeks after 10-25 Gy. Hydroxyproline per gram of dry irradiated lung was increased at 18 weeks after 15-25 Gy. Support for the hypothesis was that hydroxyproline per gram only increased after X-ray doses which caused marked extravasation of protein. There was no evidence, however, for deposition of /sup 125/I-fibrin or for a gradual increase in fibrosis corresponding to the prolonged excess of extravascular protein.

Research Organization:
Stanford Univ. School of Medicine, CA
OSTI ID:
5227733
Report Number(s):
CONF-860423-
Journal Information:
Radiat. Res.; (United States), Journal Name: Radiat. Res.; (United States) Vol. 103:1; ISSN RAREA
Country of Publication:
United States
Language:
English

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