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Distribution of sensitivity to 4-nitroquinoline 1-oxide among Japanese lymphoblastoid cell lines

Journal Article · · Bulletin of Environmental Contamination and Toxicology; (United States)
DOI:https://doi.org/10.1007/BF01688255· OSTI ID:5085842
; ;  [1];  [2]
  1. Kyushu Univ., Fukuoka (Japan)
  2. Nakamura Gakuen Coll., Fukuoka (Japan) Nakamura Junior Coll., Fukuoka (Japan)
The processes through which the UV-mimic chemical carcinogen, 4-nitroquinoline 1-oxide (4NQO), leads to the DNA lesions are well characterized in E. coli, where the formation of stable 4NQO-purine adducts is critical. The DNA excision-repair mechanisms similar to those for E. coli occur in normal human cells. Xeroderma pigmentosum (XP) is an example of a rare recessive autosomal skin disorder which is characterized biochemically as a DNA repair-deficient disease. The fluorescein diacetate (FDA) method was recently used to determine the sensitivity of lymphoblastoid cell lines 4NQO. Viable cells take up, non-fluorescent chemical, FDA and convert it to, a fluorescent molecule, fluorescein by intracellular esterases. DNA damage produced by 4NQO could be evaluated on the basis of the cell lethality by this FDA method. In the present study the authors describe the distribution of sensitivity to 4NQO among lymphoblastoid cell lines established from Japanese.
OSTI ID:
5085842
Journal Information:
Bulletin of Environmental Contamination and Toxicology; (United States), Journal Name: Bulletin of Environmental Contamination and Toxicology; (United States) Vol. 46:1; ISSN 0007-4861; ISSN BECTA
Country of Publication:
United States
Language:
English