Management of Antiphospholipid Antibody Syndrome: A Systematic Review
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March 2006 |
Toll-like receptor and antiphospholipid mediated thrombosis: in vivo studies
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October 2007 |
Refinement of Macromolecular Structures by the Maximum-Likelihood Method
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May 1997 |
Crystal structure of human β2-glycoprotein I: implications for phospholipid binding and the antiphospholipid syndrome
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November 1999 |
Annexin A2 is involved in antiphospholipid antibody-mediated pathogenic effects in vitro and in vivo
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October 2009 |
Annexin A2: biology and relevance to the antiphospholipid syndrome
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October 2008 |
Platelet adhesion to dimeric β 2 -glycoprotein I under conditions of flow is mediated by at least two receptors: glycoprotein Ib α and apolipoprotein E receptor 2′
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February 2007 |
The annexin A5-mediated pathogenic mechanism in the antiphospholipid syndrome: role in pregnancy losses and thrombosis
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March 2010 |
Coagulation and fibrinolytic activities in 2 siblings with β2-glycoprotein I deficiency
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August 2000 |
Adhesion mechanism of human beta 2-glycoprotein I to phospholipids based on its crystal structure
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October 1999 |
Patients with antiphospholipid syndrome display endothelial perturbation
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March 2010 |
Structural insights into recognition of β2-glycoprotein I by the lipoprotein receptors: Recognition of B2GPI by Lipoprotein Receptors
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July 2009 |
Role of Divalency in the High-Affinity Binding of Anticardiolipin Antibody−β 2 -Glycoprotein I Complexes to Lipid Membranes
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January 1996 |
Anti-Beta 2-Glycoprotein I Antibody Isotype and IgG Subclass in Antiphospholipid Syndrome Patients
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January 1999 |
Antiphospholipid Syndrome
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September 2009 |
Anti-phospholipid antibodies are directed against a complex antigen that includes a lipid-binding inhibitor of coagulation: beta 2-glycoprotein I (apolipoprotein H).
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June 1990 |
β2-Glycoprotein I can exist in 2 conformations: implications for our understanding of the antiphospholipid syndrome
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August 2010 |
How do antiphospholipid antibodies bind ?2-glycoprotein I?
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August 2003 |
The association between circulating antibodies against domain I of beta2-glycoprotein I and thrombosis: an international multicenter study
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November 2009 |
Toll-like receptors: another player in the pathogenesis of the anti-phospholipid syndrome
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October 2008 |
Lipoprotein Receptors in the Nervous System
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June 2002 |
Structure of an LDLR-RAP Complex Reveals a General Mode for Ligand Recognition by Lipoprotein Receptors
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April 2006 |
Platelets express three different splice variants of ApoER2 that are all involved in signaling
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July 2007 |
Antiphospholipid antibodies: Lessons from the bench
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March 2007 |
Circulating levels of β2-glycoprotein I in thrombotic disorders and in inflammation
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February 2006 |
Solution Structure of the Sixth LDL-A Module of the LDL Receptor ,
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February 2000 |
Intracellular signaling triggered by antiphospholipid antibodies in platelets and endothelial cells: a pathway to targeted therapies
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January 2004 |
Identification of Structural Mutations in the Fifth Domain of Apolipoprotein H ( 2-Glycoprotein I) Which Affect Phospholipid Binding
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February 1997 |
International consensus statement on an update of the classification criteria for definite antiphospholipid syndrome (APS)
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February 2006 |
Coot model-building tools for molecular graphics
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November 2004 |
Research around β2-glycoprotein I: A major target for antiphospholipid antibodies
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January 2005 |
Thrombolysis in antiphospholipid syndrome: Current hematologic perspectives
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February 2008 |
β2-Glycoprotein I Deficiency and the Risk of Thrombosis
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January 1992 |
β2-glycoprotein I–dependent lupus anticoagulant highly correlates with thrombosis in the antiphospholipid syndrome
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December 2004 |
Antiphospholipid syndrome: Current insights into laboratory diagnosis and pathophysiology
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January 2010 |
Anti–β2-glycoprotein I antibodies in complex with β2-glycoprotein I can activate platelets in a dysregulated manner via glycoprotein Ib-IX-V
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January 2006 |
PHENIX: a comprehensive Python-based system for macromolecular structure solution
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January 2010 |
Folding and Binding Integrity of Variants of a Prototype Ligand-Binding Module from the LDL Receptor Possessing Multiple Alanine Substitutions †
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April 2005 |
Mode of Interaction between β2GPI and Lipoprotein Receptors Suggests Mutually Exclusive Binding of β2GPI to the Receptors and Anionic Phospholipids
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March 2010 |
In vivo inhibition of antiphospholipid antibody-induced pathogenicity utilizing the antigenic target peptide domain I of β 2 -glycoprotein I: proof of concept
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May 2009 |
A hydrophobic sequence at position 313-316 (Leu-Ala-Phe-Trp) in the fifth domain of apolipoprotein H (β2-glycoprotein I) is crucial for cardiolipin binding: Binding of a hydrophobic sequence of apoH to cardiolipin
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March 2000 |
How we diagnose the antiphospholipid syndrome
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January 2009 |
Versatility in ligand recognition by LDL receptor family proteins: advances and frontiers
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August 2007 |
A deliberate approach to screening for initial crystallization conditions of biological macromolecules
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April 2003 |
Current insight into diagnostics and pathophysiology of the antiphospolipid syndrome
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March 2008 |
Structure and Physiologic Function of the Low-Density Lipoprotein Receptor
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June 2005 |
Interaction of ? 2 -glycoprotein I with members of the low density lipoprotein receptor family
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August 2006 |
Phaser crystallographic software
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July 2007 |
Anticardiolipin antibodies (ACA) directed not to cardiolipin but to a plasma protein cofactor
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June 1990 |
Solution Structure of Human and Bovine β2-Glycoprotein I Revealed by Small-angle X-ray Scattering
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August 2002 |
Dimers of β2-Glycoprotein I Increase Platelet Deposition to Collagen via Interaction with Phospholipids and the Apolipoprotein E Receptor 2′
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September 2003 |
The integration of macromolecular diffraction data
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December 2005 |
In search for a receptor for antiphospholipid antibodies on target cells
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August 2006 |
A snappy new concept for APS
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August 2010 |
PHENIX: a comprehensive Python-based system for macromolecular structure solution.
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January 2010 |
The anti-beta2-glycoprotein I activity in human anti-phospholipid syndrome sera is due to monoreactive low-affinity autoantibodies directed to epitopes located on native beta2-glycoprotein I and preserved during species' evolution.
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December 1996 |