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Impaired transport of thyroid hormones into livers of obese (ob/ob) mice

Journal Article · · American Journal of Physiology; (USA)
OSTI ID:7191998
;  [1]
  1. Michigan State Univ., Lansing (USA)

Obese (ob/ob) mice exhibit impaired hepatic thyroid hormone action that is mediated, at least in part, by a reduced nuclear 3,5,3{prime}-triiodothyronine (T{sub 3}) receptor occupancy. The possibility that lowered occupancy in obese mice may be caused by decreased transport of T{sub 3} across the hepatic plasma membrane was examined by measuring the unidirectional influx of ({sup 125}I)T{sub 3} into livers of 8- to 10-wk-old obese and lean mice using a tissue-sampling portal vein-injection technique. Influx of ({sup 125}I)thyroxine (T{sub 4}), a substrate for T{sub 4} 5{prime}-deiodinase, was also measured. Unidirectional clearance of T{sub 3} and T{sub 4} was 64 and 80% lower, respectively, in obese mice than in lean mice. Hepatic T{sub 3} and T{sub 4} uptake was nonsaturable in both lean and obese mice, suggesting that transport occurs by lipid-mediated free diffusion. Clearance of another lipid-soluble hormone, hydrocortisone, was also lower in obese mice than in lean mice. Decreased membrane permeability to the above hormones in obese mice may result from reported changes in membrane lipid composition. In conclusion, decreased hepatic thyroid hormone uptake may contribute to impaired thyroid hormone action and T{sub 3} production in livers of obese mice.

OSTI ID:
7191998
Journal Information:
American Journal of Physiology; (USA), Journal Name: American Journal of Physiology; (USA) Vol. 255:1; ISSN 0002-9513; ISSN AJPHA
Country of Publication:
United States
Language:
English

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