Skip to main content
U.S. Department of Energy
Office of Scientific and Technical Information

Mechanism of localization of /sup 99m/Tc-labeled pyrophosphate and tetracycline in infarcted myocardium

Journal Article · · J. Nucl. Med.; (United States)
OSTI ID:7187503

The gross and subcellular localizations of /suJm/Tc-labeled pyrophosphate and tetracycline in myocardial infarcts were studied in a rabbit model. Experiments utilizing double-nuclide labeling were carried out using a useful mapping technique. Concentration of the various chelates decreases in an expected manner from the center of the infarcted area toward its periphery, but it is higher near the epicardial surface than toward the endocardium. Technetium-99m-pyrophosphate is concentrated in the same infarcted areas as /sup 45/Ca ion or /sup 32/P-pyrophosphate, but to a much greater degree. The uptake is dependent on both the degree of necrosis and residual blood flow. Gel filtration experiments with rabbit serum indicate that /sup 99m/Tc-tagged pyrophosphate, tetracycline, and diphosphonate are mainly protein-bound, whereas /sup 32/P-pyrophosphate is not. Subcellular localization studies show that /sup 99m/Tc-tetracycline and /sup 99m/Tc-pyrophosphate are bound primarily to soluble protein, and only a small fraction is associated with nuclei, mitochondria, and microsomes. The uptake of technetium chelates in myocardial infarcts may be due to the formation of polynuclear complexes with denatured macromolecules rather than to the deposition of calcium in mitochondria.

Research Organization:
Tufts Univ., Boston
OSTI ID:
7187503
Journal Information:
J. Nucl. Med.; (United States), Journal Name: J. Nucl. Med.; (United States) Vol. 17:7; ISSN JNMEA
Country of Publication:
United States
Language:
English