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Hepatic handling of bile salts and protein in the rat during intrahepatic cholestasis

Journal Article · · Gastroenterology; (United States)
OSTI ID:5776794

17 alpha-Ethynyl estradiol-induced cholestasis was used to study the relationship of protein to bile salt transport in liver. The biliary secretion of horseradish peroxidase was unaltered in treated animals despite a 56% reduction in bile flow. Cytochemistry confirmed that estradiol caused no alteration in the handling of tracer. In a second study, the peak biliary secretion of (/sup 14/C)taurocholate was reduced by approximately 46% in treated animals. The kinetics of /sup 125/I-cholyglycylhistamine, a bile salt derivative, were identical to those of taurocholate in control and cholestatic animals. Taurocholate and cholylglycylhistamine secretion were markedly reduced in control animals during competition with unlabeled taurocholate. Quantitative electron microscopic autoradiography with /sup 125/I-cholylglycylhistamine revealed a high concentration of grains over the endoplasmic reticulum and the Golgi complex including associated lysosomes and vesicles. These data demonstrate that estradiol markedly inhibits bile salt transport, but not vesicular transport of horseradish peroxidase. Furthermore, estradiol may alter the movement of bile salts through these organelles.

Research Organization:
Cell Biology and Aging Section, Veterans Administration Medical Center, San Francisco, California
OSTI ID:
5776794
Journal Information:
Gastroenterology; (United States), Journal Name: Gastroenterology; (United States) Vol. 84:5; ISSN GASTA
Country of Publication:
United States
Language:
English

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