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Increased norepinephrine release during sympathetic nerve stimulation and its inhibition by adenosine in the isolated perfused kidney of spontaneously hypertensive rats

Journal Article · · Clin. Exp. Hypertens.; (United States)

The present study was performed to measure norepinephrine release during sympathetic nerve stimulation and determine the inhibitory action of adenosine on stimulus-induced release of norepinephrine in the isolated perfused kidney of WKY and SHR. Norepinephrine release during periarterial nerve stimulation was measured as total /sup 3/H-overflow since greater than 75% of total /sup 3/H-overflow was /sup 3/H-norepinephrine in both the WKY and SHR. A significantly greater increase in /sup 3/H-norepinephrine overflow was observed during periarterial nerve stimulation in SHR in comparison with WKY. Adenosine (0.3, 1.0, 3.0 and 10.0 micrograms/ml) produced dose-dependent inhibition of /sup 3/H-norepinephrine overflow elicited by periarterial nerve stimulation. However, the effect of adenosine on transmitter release was more pronounced in the SHR in that the threshold dose required to cause inhibition of stimulus-induced release of /sup 3/H-norepinephrine was smaller in the SHR. These results demonstrate that while norepinephrine release during sympathetic nerve stimulation is greater in the SHR, this finding can not be explained on the basis of a decrease in the presynaptic inhibitory action of adenosine. Therefore, the mechanism responsible for the increased release of norepinephrine in the SHR remains to be determined.

Research Organization:
Department of Pharmacology, College of Pharmacy, University of Houston, TX
OSTI ID:
5776748
Journal Information:
Clin. Exp. Hypertens.; (United States), Journal Name: Clin. Exp. Hypertens.; (United States) Vol. 5:1; ISSN CEHYD
Country of Publication:
United States
Language:
English