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Mechanism of gallium-67 accumulation in tumors

Journal Article · · J. Nucl. Med.; (United States)
OSTI ID:5019943

Neoplasms are characterized by increased perfusion, increased permeability of their capillary beds to macromolecules, and a delay in new lymphatic vessel growth. These lead to the increased entry and residency time of macromolecules in the interstitial space of tumors. Multiple factors contribute to the localization of /sup 67/Ga in tumors. Adequate blood supply is essential; at areas with no blood supply such as the necrotic center of a large tumor, there is no /sup 67/Ga accumulation. Gallium-67, mainly in the form of transferrin-67Ga complex, is delivered to the tumor through capillaries with increased permeability. In tumors, some /sup 67/Ga is taken up by tumor cells; some may also be taken up by inflammatory cells when they are present. Gallium-67 binding proteins, such as lactoferrin or ferritin, may also contribute to the accumulation and retention of /sup 67/Ga in tumors; however, their roles are less clear. The intensity of these various factors determine their relative contribution and the degree of /sup 67/Ga accumulation in tumors.56 references.

OSTI ID:
5019943
Journal Information:
J. Nucl. Med.; (United States), Journal Name: J. Nucl. Med.; (United States) Vol. 7; ISSN JNMEA
Country of Publication:
United States
Language:
English