Initial HIV-1 Antigen-Specific CD8+ T Cells in Acute HIV-1 Infection Inhibit Transmitted/Founder Virus Replication
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Temporal Dynamics of CD8+ T Cell Effector Responses during Primary HIV Infection
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Augmentation of HIV-specific T cell function by immediate treatment of hyperacute HIV-1 infection
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Mean-field computational approach to HIV dynamics on a fitness landscape
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Genetic identity, biological phenotype, and evolutionary pathways of transmitted/founder viruses in acute and early HIV-1 infection
- S., Perelson, Alan; Christina, Ochsenbauer-Jambor,; C., Kappas, John
-
The University of North Carolina at Chapel Hill University Libraries
https://doi.org/10.17615/1n1a-3d64
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Genetic identity, biological phenotype, and evolutionary pathways of transmitted/founder viruses in acute and early HIV-1 infection
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Innate Immune Activity Correlates with CD4 T Cell-Associated HIV-1 DNA Decline during Latency-Reversing Treatment with Panobinostat
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Harnessing CD8+ T Cells Under HIV Antiretroviral Therapy
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Vertical T cell immunodominance and epitope entropy determine HIV-1 escape
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Transmitted/Founder and Chronic Subtype C HIV-1 Use CD4 and CCR5 Receptors with Equal Efficiency and Are Not Inhibited by Blocking the Integrin α4β7
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Early Low-Titer Neutralizing Antibodies Impede HIV-1 Replication and Select for Virus Escape
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Genetic identity, biological phenotype, and evolutionary pathways of transmitted/founder viruses in acute and early HIV-1 infection
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Estimating the mutational fitness effects distribution during early HIV infection
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Discrimination Between Human Leukocyte Antigen Class I-Bound and Co-Purified HIV-Derived Peptides in Immunopeptidomics Workflows
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Terminal Effector CD8 T Cells Defined by an IKZF2 + IL-7R − Transcriptional Signature Express FcγRIIIA, Expand in HIV Infection, and Mediate Potent HIV-Specific Antibody-Dependent Cellular Cytotoxicity
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Implications of CTL-Mediated Killing of HIV-Infected Cells during the Non-Productive Stage of Infection
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In vivo mutation rates and the landscape of fitness costs of HIV-1
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Clonal Expansion of Human Immunodeficiency Virus–Infected Cells and Human Immunodeficiency Virus Persistence During Antiretroviral Therapy
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Rare HIV-1 transmitted/founder lineages identified by deep viral sequencing contribute to rapid shifts in dominant quasispecies during acute and early infection
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