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K-Cl transport systems in rabbit renal basolateral membrane vesicles

Journal Article · · Am. J. Physiol.; (United States)
OSTI ID:6804781

The transport pathways for chloride in basolateral membrane vesicles from the rabbit renal cortex were investigated. /sup 36/Cl uptake was stimulated by the presence of potassium in the uptake media compared with sodium of N-methyl-D-glucamine. In addition, potassium (/sup 86/Rb) uptake was stimulated more by chloride than by nitrate or gluconate. Neither of these processes was further stimulated by potassium gradients plus valinomycin, suggesting the presence of an electrically neutral K-Cl cotransport system. A magnesium-induced chloride conductance was also found in the basolateral membrane vesicles. In the absence of magnesium, the chloride conductance was low; valinomycin and an inwardly directed potassium gradient did not stimulated /sup 36/Cl uptake, anthracene-9-carboxylic acid did not inhibit /sup 36/Cl uptake, and valinomycin did not stimulated chloride-dependent /sup 86/Rb uptake. However, in the presence of 1 mM magnesium, opposite results were obtained; valinomycin and an inwardly directed potassium gradient stimulated /sup 36/Cl uptake, anthracene-9-carboxylic acid inhibited /sup 36/Cl uptake, and valinomycin stimulated chloride-dependent /sup 86/Rb uptake. Therefore, an electrically neutral K-Cl cotransport and magnesium-induced chloride conductance were found in renal cortial basolateral membrane vesicles prepared from the rabbit renal cortex.

Research Organization:
Veterans Administration Medical Center, San Francisco (USA)
OSTI ID:
6804781
Journal Information:
Am. J. Physiol.; (United States), Journal Name: Am. J. Physiol.; (United States) Vol. 252:5; ISSN AJPHA
Country of Publication:
United States
Language:
English

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