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Role of bone marrow cells in the growth inhibition of transplanted methylcholanthrene-induced sarcoma (MCA). [/sup 137/Cs]

Conference · · Transplant. Soc., Int. Cong., Proc.; (United States)
OSTI ID:6525299

The operation of various antitumor mechanisms in vivo remains largely unknown. The presence of specific sensitized cells in the spleen and lymph node of tumor-bearing and tumor-immune animals has been demonstrated. Such cells are more effectively revealed by secondary sensitization of the splenocytes of tumor-immune animals in vitro by reexposing them to the tumor. Tumor-immune in vitro sensitized splenocytes (ISS) are highly effective in retarding the in vivo growth of the sensitizing tumor when they are transplated together with tumor cells into normal recipient mice. However, if the same cell transfer is made into lethally irradiated mice, the tumor will grow rapidly in 100% of recipients. The objective of the present study is to explore this phenomenon and to determine the origin of the host cells that are capable of collaborating with tumor-immune in vitro sensitized splenocytes in the regulation of tumor growth. We designed experiments to test whether the host cells involved in tumor growth regulation are derived from the spleen, thymus, or the bone marrow and set out to determine whether or not collaboration of the host cells with ISS was specific as far as the sensitizing tumor was concerned.

Research Organization:
Univ. of Washington, Seattle
DOE Contract Number:
EY-76-S-06-2225
OSTI ID:
6525299
Journal Information:
Transplant. Soc., Int. Cong., Proc.; (United States), Journal Name: Transplant. Soc., Int. Cong., Proc.; (United States) Vol. 13:1; ISSN TICPB
Country of Publication:
United States
Language:
English

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