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Cyclic AMP and CaS -activated K transport in a human colonic epithelial cell line

Journal Article · · J. Biol. Chem.; (United States)
OSTI ID:6292117

Addition of either vasoactive intestinal peptide (VIP) or the CaS ionophore, A23187, to confluent monolayers of the T84 epithelial cell line derived from a human colon carcinoma increased the rate of YWRb or USK efflux from preloaded cells. The effect of A23187 required extracellular CaS , while that of VIP correlated with its known effect on cyclic AMP production. Other agents which increased cyclic AMP production or mimicked its effect also increased YWRb efflux. VIP- or A23187-stimulated efflux was inhibited by 5 mM BaS or 1 mM quinidine, but not by 20 mM tetraethylammonium, 4 mM 4-aminopyridine, or 1 microM apamin. Under appropriate conditions, VIP and A23187 also increased the rate of YWRb or USK uptake. Stimulation of the initial rate of uptake by either agent required high intracellular K and was not markedly affected by the imposition of transcellular pH gradients. The effect of A23187, but not VIP or dibutyryl cyclic AMP, was refractory to depletion of cellular energy stores. A23187-stimulated uptake was not significantly affected by anion substitution, however, stimulation of uptake by VIP required the presence of a permeant anion. This result may be due to the simultaneous activation of a cyclic AMP-dependent Cl transport system. The kinetics of both VIP- and A23187-stimulated uptake and efflux were consistent with a channel-rather than a carrier-mediated K transport mechanism. The results also suggest that cyclic AMP and CaS may activate two different kinds of K transport systems. Finally, both transport systems have been localized to the basolateral membrane of T84 monolayers, a result compatible with their possible regulatory role in hormone-activated electrogenic Cl secretion.

Research Organization:
Univ. of California, San Diego
OSTI ID:
6292117
Journal Information:
J. Biol. Chem.; (United States), Journal Name: J. Biol. Chem.; (United States) Vol. 26; ISSN JBCHA
Country of Publication:
United States
Language:
English

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