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Correction of a nucleotide-excision-repair mutation by human chromosome 19 in hamster-human hybrid cells

Journal Article · · Somatic Cell Mol. Gen.; (United States)
DOI:https://doi.org/10.1007/BF01534738· OSTI ID:5554941
A UV-sensitive mutant line of CHO cells, UV20, was shown to be phenotypically corrected to resistance by fusion with human lymphocytes or fibroblasts. Only human chromosome 19 correlated with the DNA repair phenotype of resistant hybrid clones and their resistant or sensitive subclones. This study demonstrates the mapping of a human repair gene by direct selection of complementing hybrids in the presence of a DNA-damaging agent (mitomycin C).
Research Organization:
Lawrence Livermore National Lab., CA
OSTI ID:
5554941
Journal Information:
Somatic Cell Mol. Gen.; (United States), Journal Name: Somatic Cell Mol. Gen.; (United States) Vol. 11:1; ISSN SCMGD
Country of Publication:
United States
Language:
English