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X-linked ichthyosis without STS deficiency: Clinical, genetical, and molecular studies

Journal Article · · American Journal of Medical Genetics
; ; ;  [1]
  1. Istituto di Genetica Molecolare del, Trieste (Italy); and others
We report on a Sardinian pedigree with congenital ichthyosis associated with normal levels of steroid sulfatase and a normal molecular pattern, as detectable with a cDNA probe for the steroid sulfatase (STS) gene. Though the pattern of transmission of the disease is consistent with X-linked recessive inheritance, this form of ichthyosis was found to segregate independently of genetic polymorphisms detected by probes of the region Xp22.3, where the STS locus has been mapped. The search for close genetic linkages with other polymorphic markers scattered along the entire X chromosome has so far been fruitless. For the time being, the main conclusion derived from these data is that STS deficiency is not a sine qua non for X-linked ichthyosis which may also result from a mutational event at an X-chromosomal site genetically unlinked to the STS locus. 16 refs., 4 figs.
Sponsoring Organization:
USDOE
OSTI ID:
443803
Journal Information:
American Journal of Medical Genetics, Journal Name: American Journal of Medical Genetics Journal Issue: 2 Vol. 59; ISSN 0148-7299; ISSN AJMGDA
Country of Publication:
United States
Language:
English

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