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microRNA-608 inhibits human hepatocellular carcinoma cell proliferation via targeting the BET family protein BRD4

Journal Article · · Biochemical and Biophysical Research Communications
 [1];  [2]; ;  [3];  [1];  [1]
  1. Liver and Cholecyst Center, First Affiliated Hospital of Nanjing Medical University, Nanjing (China)
  2. Nursing College, Nanjing University of Chinese Medicine, Nanjing (China)
  3. Department of General Surgery, Affiliated Hospital of Nanjing University of Chinese Medicine, Nanjing (China)

Highlights: • miR-608 expression downregulates BRD4, inhibiting human HCC cell proliferation. • The miR-608 inhibitor increases BRD4 expression to promote HepG2 cell proliferation. • BRD4 is the primary target gene of miR-608 in HepG2 cells. • miR-608 expression inhibits HepG2 xenograft growth in SCID mice. • BRD4 upregulation correlates with miR-608 downregulation in human HCC tissues. Over-expression of the bromodomain and extraterminal (BET) family protein BRD4 is associated with hepatocellular carcinoma (HCC) progression. In the present study, we indentified a novel putative anti-BRD4 microRNA: microRNA-608 (“miR-608”). In HepG2 cells and primary human HCC cells, over-expression of miR-608, using a lentiviral construct, induced BRD4 downregulation and proliferation inhibition. Conversely, transfection of the miR-608 inhibitor increased BRD4 expression to promote HepG2 cell proliferation. Our results suggest that BRD4 is the primary target gene of miR-608 in HepG2 cells. shRNA-mediated knockdown or CRSIPR/Cas9-mediated knockout of BRD4 mimicked and overtook miR-608's actions in HepG2 cells. Furthermore, introduction of a 3′-untranslated region (3′-UTR) mutant BRD4 (UTR-A1718G) blocked miR-608-induced c-Myc downregulation and proliferation inhibition in HepG2 cells. In vivo, HepG2 xenograft tumor growth was significantly inhibited after expressing miR-608 or BRD4 CRSIPR/Cas9-KO construct. Importantly, BRD4 mRNA was upregulated in human HCC tissues, which was correlated with downregulation of miR-608. Together, we conclude that miR-608 inhibits HCC cell proliferation possibly via targeting BET family protein BRD4.

OSTI ID:
23125052
Journal Information:
Biochemical and Biophysical Research Communications, Journal Name: Biochemical and Biophysical Research Communications Journal Issue: 4 Vol. 501; ISSN 0006-291X; ISSN BBRCA9
Country of Publication:
United States
Language:
English

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