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High dose of plasmid IL-15 inhibits immune responses in an influenza non-human primates immunogenicity model

Journal Article · · Virology
OSTI ID:21357543
; ; ; ;  [1];  [2]; ;  [3];  [4];  [2];  [1]
  1. Department of Pathology and Laboratory Medicine, University of Pennsylvania School of Medicine, 505 SCL, 422 Curie Blvd. Philadelphia, PA 19104 (United States)
  2. VGX Pharmaceuticals, Inc., The Woodlands, TX 77381 (United States)
  3. Research Section, Bioqual, Rockville, MD 20850 (United States)
  4. Drexel University College of Medicine, Philadelphia, PA 19102 (United States)

Interleukin (IL)-15, is a cytokine that is important for the maintenance of long-lasting, high-avidity T cell response to invading pathogens and has, therefore, been used in vaccine and therapeutic platforms as an adjuvant. In addition to pure protein delivery, plasmids encoding the IL-15 gene have been utilized. However, it is critical to determine the appropriate dose to maximize the adjuvanting effects. We immunized rhesus macaques with different doses of IL-15 expressing plasmid in an influenza non-human primate immunogenicity model. We found that co-immunization of rhesus macaques with a Flu DNA-based vaccine and low doses of plasmid encoding macaque IL-15 enhanced the production of IFN-gamma (0.5 mg) and the proliferation of CD4{sup +} and CD8{sup +} T cells, as well as T{sub CM} levels in proliferating CD8{sup +} T cells (0.25 mg). Whereas, high doses of IL-15 (4 mg) decrease the production of IFN-gamma and the proliferation of CD4{sup +} and CD8{sup +} T cells and T{sub CM} levels in the proliferating CD4{sup +} and CD8{sup +} T cells. In addition, the data of hemagglutination inhibition (HI) antibody titer suggest that although not significantly different, there appears to be a slight increase in antibodies at lower doses of IL-15. Importantly, however, the higher doses of IL-15 decrease the antibody levels significantly. This study demonstrates the importance of optimizing DNA-based cytokine adjuvants.

OSTI ID:
21357543
Journal Information:
Virology, Journal Name: Virology Journal Issue: 1 Vol. 393; ISSN VIRLAX; ISSN 0042-6822
Country of Publication:
United States
Language:
English