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Characterization of a novel oncogenic K-ras mutation in colon cancer

Journal Article · · Biochemical and Biophysical Research Communications
 [1];  [2];  [3];  [2];  [4];  [4]
  1. Molecular Diagnosis and Cancer Prevention Division, Saitama Cancer Center, 818 Komuro Ina, Kitaadachigun, Saitama 362-0806 (Japan) and Research Institute for Clinical Oncology, Saitama Cancer Center, 818 Komuro Ina, Kitaadachigun, Saitama 362-0806 (Japan)
  2. Molecular Diagnosis and Cancer Prevention Division, Saitama Cancer Center, 818 Komuro Ina, Kitaadachigun, Saitama 362-0806 (Japan)
  3. Gastroenterology Division, Saitama Cancer Center, 818 Komuro Ina, Kitaadachigun, Saitama 362-0806 (Japan)
  4. Research Institute for Clinical Oncology, Saitama Cancer Center, 818 Komuro Ina, Kitaadachigun, Saitama 362-0806 (Japan)
Activating mutations of RAS are frequently observed in subsets of human cancers, indicating that RAS activation is involved in tumorigenesis. Here, we identified and characterized a novel G to T transversion mutation of the K-ras gene at the third position of codon 19 (TTG) which substituted phenylalanine for leucine in 3 primary colon carcinomas. Biological and biochemical activity was examined using transformed NIH3T3 cells expressing mutant or wild-type K-ras. Transformants harboring the K-ras mutation at codon 19 showed proliferative capacity under serum-starved conditions, less contact inhibition, anchorage-independent growth, tumorigenicity in nude mice and elevation of active Ras-GTP levels. These results indicated that this novel mutation possesses high oncogenic activity.
OSTI ID:
20857968
Journal Information:
Biochemical and Biophysical Research Communications, Journal Name: Biochemical and Biophysical Research Communications Journal Issue: 3 Vol. 352; ISSN BBRCA9; ISSN 0006-291X
Country of Publication:
United States
Language:
English

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