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NS1 DNA vaccination protects against Zika infection through T cell–mediated immunity in immunocompetent mice

Journal Article · · Science Advances
 [1];  [1];  [1];  [2];  [2];  [3];  [2];  [1];  [1];  [4];  [4];  [4];  [5];  [4];  [6];  [7];  [1]
  1. Univ. of Adelaide, SA (Australia); Basil Hetzel Inst. for Translational Health Research, Adelaide, SA (Australia)
  2. Harvard Medical School, Boston, MA (United States)
  3. Univ. of South Australia, Adelaide, SA (Australia); QIMR Berghofer Medical Research Inst., Brisbane, QLD (Australia); Australian Infectious Diseases Research Centre, Brisbane, QLD (Australia)
  4. Univ. of Adelaide, SA (Australia)
  5. Finders Univ., Adelaide, SA (Australia)
  6. Univ. of South Australia, Adelaide, SA (Australia); Univ. of Adelaide, SA (Australia)
  7. Harvard Medical School, Boston, MA (United States); Ragon Inst. of MGH, MIT, and Harvard, Cambridge, MA (United States)

The causal association of Zika virus (ZIKV) with microcephaly, congenital malformations in infants, and Guillain-Barré syndrome in adults highlights the need for effective vaccines. Thus far, efforts to develop ZIKV vaccines have focused on the viral envelope. ZIKV NS1 as a vaccine immunogen has not been fully explored, although it can circumvent the risk of antibody-dependent enhancement of ZIKV infection, associated with envelope antibodies. Here, we describe a novel DNA vaccine encoding a secreted ZIKV NS1, that confers rapid protection from systemic ZIKV infection in immunocompetent mice. We identify novel NS1 T cell epitopes in vivo and show that functional NS1-specific T cell responses are critical for protection against ZIKV infection. We demonstrate that vaccine-induced anti-NS1 antibodies fail to confer protection in the absence of a functional T cell response. This highlights the importance of using NS1 as a target for T cell–based ZIKV vaccines.

Research Organization:
Lawrence Berkeley National Laboratory (LBNL), Berkeley, CA (United States)
Sponsoring Organization:
USDOE Office of Science (SC)
Grant/Contract Number:
AC02-05CH11231
OSTI ID:
1591849
Journal Information:
Science Advances, Journal Name: Science Advances Journal Issue: 12 Vol. 5; ISSN 2375-2548
Publisher:
AAASCopyright Statement
Country of Publication:
United States
Language:
English

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Cited By (2)

Two Is Better Than One: Evidence for T-Cell Cross-Protection Between Dengue and Zika and Implications on Vaccine Design journal March 2020
Zika Virus Inhibits IFN-α Response by Human Plasmacytoid Dendritic Cells and Induces NS1-Dependent Triggering of CD303 (BDCA-2) Signaling journal October 2020

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