skip to main content

Title: Seasonal relationship of sudden infant death syndrome and environmental pollutants

Evidence that chronic hypoxia precedes death from sudden infant death syndrome (SIDS) is mounting. Prolonged exposure to moderate levels of pollutants could be a contributing factor to hypoxia. Levels of carbon monoxide (CO), sulfur dioxide (SO/sub 2/), nitrogen dioxide (NO/sub 2/) and hydrocarbons (HC) are highest in the winter when incidence of SIDS is increased. SIDS cases in Los Angeles County were correlated with daily mean levels of these pollutants, temperature, barometric pressure and monthly lead levels with the aid of time series analyses. Peaks in CO, SO/sub 2/, NO/sub 2/, HC and lead preceded the seasonal increase in SIDS by seven weeks. Theoretical considerations, such as the hypoxia-inducing effects of CO, support the hypothesis that this temporal relation has functional significance. The role of pollution levels as a predisposing factor in risk for SIDS cannot be summarily dismissed.
Authors:
 [1] ; ; ;
  1. (Los Angeles County/Univ. of Southern California, Los Angeles)
Publication Date:
OSTI Identifier:
6447921
Resource Type:
Journal Article
Resource Relation:
Journal Name: Am. J. Epidemiol.; (United States); Journal Volume: 113:6
Country of Publication:
United States
Language:
English
Subject:
63 RADIATION, THERMAL, AND OTHER ENVIRON. POLLUTANT EFFECTS ON LIVING ORGS. AND BIOL. MAT.; 54 ENVIRONMENTAL SCIENCES; 59 BASIC BIOLOGICAL SCIENCES; AIR POLLUTION; SEASONAL VARIATIONS; CARBON MONOXIDE; BIOLOGICAL EFFECTS; DEATH; EPIDEMIOLOGY; HYDROCARBONS; LEAD; NITROGEN DIOXIDE; SULFUR DIOXIDE; AMBIENT TEMPERATURE; ANOXIA; INFANTS; AGE GROUPS; CARBON COMPOUNDS; CARBON OXIDES; CHALCOGENIDES; CHILDREN; ELEMENTS; METALS; NITROGEN COMPOUNDS; NITROGEN OXIDES; ORGANIC COMPOUNDS; OXIDES; OXYGEN COMPOUNDS; POLLUTION; SULFUR COMPOUNDS; SULFUR OXIDES; VARIATIONS 560306* -- Chemicals Metabolism & Toxicology-- Man-- (-1987); 500200 -- Environment, Atmospheric-- Chemicals Monitoring & Transport-- (-1989); 550100 -- Behavioral Biology