Ecdysone signaling at metamorphosis triggers apoptosis of Drosophila abdominal muscles

doi:10.1016/j.ydbio.2013.08.029

Title: Ecdysone signaling at metamorphosis triggers apoptosis of Drosophila abdominal muscles

Full Record
Other Related Research

Authors:: Zirin, Jonathan; Cheng, Daojun; Dhanyasi, Nagaraju; Cho, Julio; Dura, Jean-Maurice; VijayRaghavan, Krishnaswamy; Perrimon, Norbert

Publication Date:: 2013-11-01

Sponsoring Org.:: USDOE Office of Science (SC), Nuclear Physics (NP) (SC-26)

OSTI Identifier:: 1209796

Grant/Contract Number:: 5F32GM082174-02

Resource Type:: Published Article

Journal Name:: Developmental Biology

Additional Journal Information:: Journal Volume: 383; Journal Issue: 2; Journal ID: ISSN 0012-1606

Publisher:: Elsevier

Country of Publication:: United States

Language:: English

Citation Formats


                    Zirin, Jonathan, Cheng, Daojun, Dhanyasi, Nagaraju, Cho, Julio, Dura, Jean-Maurice, VijayRaghavan, Krishnaswamy, and Perrimon, Norbert. Ecdysone signaling at metamorphosis triggers apoptosis of Drosophila abdominal muscles.  United States: N. p., 2013. 
        Web.  doi:10.1016/j.ydbio.2013.08.029.

Copy to clipboard


                    Zirin, Jonathan, Cheng, Daojun, Dhanyasi, Nagaraju, Cho, Julio, Dura, Jean-Maurice, VijayRaghavan, Krishnaswamy, & Perrimon, Norbert. Ecdysone signaling at metamorphosis triggers apoptosis of Drosophila abdominal muscles.  United States.  doi:10.1016/j.ydbio.2013.08.029.

Copy to clipboard


                    Zirin, Jonathan, Cheng, Daojun, Dhanyasi, Nagaraju, Cho, Julio, Dura, Jean-Maurice, VijayRaghavan, Krishnaswamy, and Perrimon, Norbert. Fri .  
        "Ecdysone signaling at metamorphosis triggers apoptosis of Drosophila abdominal muscles".  United States.  doi:10.1016/j.ydbio.2013.08.029.

Copy to clipboard


                    
@article{osti_1209796,

  title        = {Ecdysone signaling at metamorphosis triggers apoptosis of Drosophila abdominal muscles},

  author       = {Zirin, Jonathan and Cheng, Daojun and Dhanyasi, Nagaraju and Cho, Julio and Dura, Jean-Maurice and VijayRaghavan, Krishnaswamy and Perrimon, Norbert},

  abstractNote = {},

  doi          = {10.1016/j.ydbio.2013.08.029},

  journal      = {Developmental Biology},

  number       = 2,

  volume       = 383,

  place        = {United States},

  year         = {2013},

  month        = {11}

}

Copy to clipboard

Journal Article:

Free Publicly Available Full Text

Publisher's Version of Record
DOI: 10.1016/j.ydbio.2013.08.029

Other availability

Search WorldCat to find libraries that may hold this journal

Citation Metrics:

Cited by: 11 works

Citation information provided by
Web of Science

Save / Share:

Export Metadata

Save to My Library

Similar Records in DOE PAGES and OSTI.GOV collections:

Pre-mRNA splicing repression triggers abiotic stress signaling in plants

Journal Article Ling, Yu ; Alshareef, Sahar ; Butt, Haroon ; ... - The Plant Journal
Cited by 7
DOI: 10.1111/tpj.13383
Newly synthesized quinazolinone HMJ-38 suppresses angiogenetic responses and triggers human umbilical vein endothelial cell apoptosis through p53-modulated Fas/death receptor signaling

Journal Article Chiang, Jo-Hua ; Yang, Jai-Sing ; Lu, Chi-Cheng ; ... - Toxicology and Applied Pharmacology

The current study aims to investigate the antiangiogenic responses and apoptotic death of human umbilical vein endothelial cells (HUVECs) by a newly synthesized compound named 2-(3′-methoxyphenyl)-6-pyrrolidinyl-4-quinazolinone (HMJ-38). This work attempted to not only explore the effects of angiogenesis on in vivo and ex vivo studies but also hypothesize the implications for HUVECs (an ideal cell model for angiogenesis in vitro) and further undermined apoptotic experiments to verify the underlying molecular signaling by HMJ-38. Our results demonstrated that HMJ-38 significantly inhibited blood vessel growth and microvessel formation by the mouse Matrigel plug assay of angiogenesis, and the suppression of microsprouting frommore »« less
DOI: 10.1016/J.TAAP.2013.03.007
Aconitine-induced Ca{sup 2+} overload causes arrhythmia and triggers apoptosis through p38 MAPK signaling pathway in rats

Journal Article Sun, Gui-bo ; Sun, Hong ; Meng, Xiang-bao ; ... - Toxicology and Applied Pharmacology

Aconitine is a major bioactive diterpenoid alkaloid with high content derived from herbal aconitum plants. Emerging evidence indicates that voltage-dependent Na{sup +} channels have pivotal roles in the cardiotoxicity of aconitine. However, no reports are available on the role of Ca{sup 2+} in aconitine poisoning. In this study, we explored the importance of pathological Ca{sup 2+} signaling in aconitine poisoning in vitro and in vivo. We found that Ca{sup 2+} overload lead to accelerated beating rhythm in adult rat ventricular myocytes and caused arrhythmia in conscious freely moving rats. To investigate effects of aconitine on myocardial injury, we performed cytotoxicitymore »« less
DOI: 10.1016/J.TAAP.2014.05.005
Dynamics of Inductive ERK Signaling in the Drosophila Embryo

Journal Article Lim, Bomyi ; Dsilva, Carmeline J. ; Levario, Thomas J. ; ... - Current Biology
Cited by 20
DOI: 10.1016/j.cub.2015.05.039
Multiple Paternity Benefits Female Marbled Salamanders by Increasing Survival of Progeny to Metamorphosis

Journal Article Croshaw, Dean A. ; Pechmann, Joseph H. K. ; Glenn, Travis C. ; ... - Ethology
Cited by 1
DOI: 10.1111/eth.12597

Similar Records