Abstract
This paper describes a female patient who had been exposed at the age of 26 to the atomic bomb at a distance of 1.3 km from the bombed area. She was pointed out to have anemia and the disturbance of the liver in 1961. In 1963, she had iron deficiency, hypoleucocytosis, and hyper-..gamma.. globulinemia, and showed a high value of serum colloids. In 1969, she had more frequently anemia together with hyper ..gamma..-globulinemia. The erythrocyte sedimentation rate and serum colloids were still high. The positive reaction to the LE cells began to appear. The liver biopsy findings showed proliferation of Kupper cells in the lobule, decidual degeneration of some liver cells, infiltration of many round cells in the Glisson's sheath. Therefore, she was diagnosed as having pure red cell aplasia and lupoid hepatitis. However, in 1970 she was suspected of having pulmonary valvular insufficiency caused by pulmonary hypertension in spite of the continuous steroid therapy. In 1974, she died of jundice, hyper-..gamma..-globulinemia which accompanied urea in the blood and the increased values of nitrogen and NH/sub 3/. This patient was considered to be interested on the assumption of the relationships between exposure to the atomic bomb and disturbance of immunity.
Citation Formats
Toyoda, N, Matsukuma, G, Uchiyama, E, Tsunoo, S, and Anpi, S.
Patient with pure red cell aplasia who was exposed to the atomic bomb near the site of blast.
Japan: N. p.,
1976.
Web.
Toyoda, N, Matsukuma, G, Uchiyama, E, Tsunoo, S, & Anpi, S.
Patient with pure red cell aplasia who was exposed to the atomic bomb near the site of blast.
Japan.
Toyoda, N, Matsukuma, G, Uchiyama, E, Tsunoo, S, and Anpi, S.
1976.
"Patient with pure red cell aplasia who was exposed to the atomic bomb near the site of blast."
Japan.
@misc{etde_7324002,
title = {Patient with pure red cell aplasia who was exposed to the atomic bomb near the site of blast}
author = {Toyoda, N, Matsukuma, G, Uchiyama, E, Tsunoo, S, and Anpi, S}
abstractNote = {This paper describes a female patient who had been exposed at the age of 26 to the atomic bomb at a distance of 1.3 km from the bombed area. She was pointed out to have anemia and the disturbance of the liver in 1961. In 1963, she had iron deficiency, hypoleucocytosis, and hyper-..gamma.. globulinemia, and showed a high value of serum colloids. In 1969, she had more frequently anemia together with hyper ..gamma..-globulinemia. The erythrocyte sedimentation rate and serum colloids were still high. The positive reaction to the LE cells began to appear. The liver biopsy findings showed proliferation of Kupper cells in the lobule, decidual degeneration of some liver cells, infiltration of many round cells in the Glisson's sheath. Therefore, she was diagnosed as having pure red cell aplasia and lupoid hepatitis. However, in 1970 she was suspected of having pulmonary valvular insufficiency caused by pulmonary hypertension in spite of the continuous steroid therapy. In 1974, she died of jundice, hyper-..gamma..-globulinemia which accompanied urea in the blood and the increased values of nitrogen and NH/sub 3/. This patient was considered to be interested on the assumption of the relationships between exposure to the atomic bomb and disturbance of immunity.}
journal = []
volume = {29:3}
journal type = {AC}
place = {Japan}
year = {1976}
month = {Mar}
}
title = {Patient with pure red cell aplasia who was exposed to the atomic bomb near the site of blast}
author = {Toyoda, N, Matsukuma, G, Uchiyama, E, Tsunoo, S, and Anpi, S}
abstractNote = {This paper describes a female patient who had been exposed at the age of 26 to the atomic bomb at a distance of 1.3 km from the bombed area. She was pointed out to have anemia and the disturbance of the liver in 1961. In 1963, she had iron deficiency, hypoleucocytosis, and hyper-..gamma.. globulinemia, and showed a high value of serum colloids. In 1969, she had more frequently anemia together with hyper ..gamma..-globulinemia. The erythrocyte sedimentation rate and serum colloids were still high. The positive reaction to the LE cells began to appear. The liver biopsy findings showed proliferation of Kupper cells in the lobule, decidual degeneration of some liver cells, infiltration of many round cells in the Glisson's sheath. Therefore, she was diagnosed as having pure red cell aplasia and lupoid hepatitis. However, in 1970 she was suspected of having pulmonary valvular insufficiency caused by pulmonary hypertension in spite of the continuous steroid therapy. In 1974, she died of jundice, hyper-..gamma..-globulinemia which accompanied urea in the blood and the increased values of nitrogen and NH/sub 3/. This patient was considered to be interested on the assumption of the relationships between exposure to the atomic bomb and disturbance of immunity.}
journal = []
volume = {29:3}
journal type = {AC}
place = {Japan}
year = {1976}
month = {Mar}
}