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Thyroid dosimetry after the Chernobyl accident and thyroid cancer in iodine deficient areas

Abstract

Of the radionuclides generated from 235-U and 239-Pu in a core of the nuclear reactor, radioiodines particularly 131-I, is the most significant in view of its huge quantities, easy dispersion and cumulation in the human thyroid in case of a nuclear accident. After nuclear accident in Chernobyl 20-50 million Ci of 131-I was released. Depending on the dose absorbed to the thyroid, 131-I can cause a late appearance of a thyroid nodule or cancer and/or thyroid destruction leading to hypothyroidism. Thyroid irradiation may origin from two sources: external cumulative radiation mainly of gamma type and internal related to 131-I cumulation. So far most information on the risk factors of the thyroid cancer due to is related to from external radiation, but there is no scientific basis to believe that internal radiation cannot induce the thyroid cancer. Thyroid dosimetry after Chernobyl accident in near and far field is essential for calculation of the thyroid cancer risk coefficient due to radiation. 1 tab.
Authors:
Szybinski, Z [1] 
  1. Jagiellonian Univ., Cracow (Poland). Dept. of Endocrinology
Publication Date:
Aug 01, 1996
Product Type:
Conference
Report Number:
IAEA-TECDOC-893; CONF-9405381-
Reference Number:
SCA: 560101; 560161; 560151; 570100; PA: AIX-28:017925; EDB-97:029791; SN: 97001735766
Resource Relation:
Conference: International symposium on radiodosimetry and preventive measures in the event of a nuclear accident, Cracow (Poland), 26-28 May 1994; Other Information: PBD: Aug 1996; Related Information: Is Part Of Radiodosimetry and preventive measures in the event of a nuclear accident. Proceedings of an international symposium; PB: 119 p.
Subject:
56 BIOLOGY AND MEDICINE, APPLIED STUDIES; 57 HEALTH AND SAFETY; THYROID; DOSIMETRY; IODINE 131; BIOLOGICAL LOCALIZATION; BIOLOGICAL RADIATION EFFECTS; CARCINOGENESIS; CHERNOBYLSK-4 REACTOR; HYPOTHYROIDISM; RADIATION DOSES; REACTOR ACCIDENTS
OSTI ID:
429924
Research Organizations:
International Atomic Energy Agency, Vienna (Austria); Polish Society of Nuclear Medicine (Poland)
Country of Origin:
IAEA
Language:
English
Other Identifying Numbers:
Journal ID: ISSN 1011-4289; Other: ON: DE97614107; TRN: XA9643026017925
Availability:
INIS; OSTI as DE97614107
Submitting Site:
INIS
Size:
pp. 9-11
Announcement Date:
Feb 24, 1997

Citation Formats

Szybinski, Z. Thyroid dosimetry after the Chernobyl accident and thyroid cancer in iodine deficient areas. IAEA: N. p., 1996. Web.
Szybinski, Z. Thyroid dosimetry after the Chernobyl accident and thyroid cancer in iodine deficient areas. IAEA.
Szybinski, Z. 1996. "Thyroid dosimetry after the Chernobyl accident and thyroid cancer in iodine deficient areas." IAEA.
@misc{etde_429924,
title = {Thyroid dosimetry after the Chernobyl accident and thyroid cancer in iodine deficient areas}
author = {Szybinski, Z}
abstractNote = {Of the radionuclides generated from 235-U and 239-Pu in a core of the nuclear reactor, radioiodines particularly 131-I, is the most significant in view of its huge quantities, easy dispersion and cumulation in the human thyroid in case of a nuclear accident. After nuclear accident in Chernobyl 20-50 million Ci of 131-I was released. Depending on the dose absorbed to the thyroid, 131-I can cause a late appearance of a thyroid nodule or cancer and/or thyroid destruction leading to hypothyroidism. Thyroid irradiation may origin from two sources: external cumulative radiation mainly of gamma type and internal related to 131-I cumulation. So far most information on the risk factors of the thyroid cancer due to is related to from external radiation, but there is no scientific basis to believe that internal radiation cannot induce the thyroid cancer. Thyroid dosimetry after Chernobyl accident in near and far field is essential for calculation of the thyroid cancer risk coefficient due to radiation. 1 tab.}
place = {IAEA}
year = {1996}
month = {Aug}
}