Abstract
Widespread use of organotins has caused increasing amounts to be released into the environment. The most important non-pesticidal route of entry of organotins into the environment is through leaching of organotin-stabilized PVC in water, and the use in antifouling agents, resulting in the introduction of organotin into the aquatic environment. Data are available regarding the detection of butyltins and phenyltins in aquatic marine organisms and marine products. Food chain bioamplification of butyltin in oysters, mud crabs, marine mussels, chinook salmons, dolphins, tunas, and sharks and of phenyltin in carps and horseshoe crabs has been reported. These findings indicate that organotins accumulate in the food chain and are bioconcentrated, and that humans can be exposed to organotins via seafood. The levels of organotin compounds in seafood are not considered to be sufficiently high to affect human health. However, Belfroid et al. (2000) noted that more research on residual TBT levels in seafood was needed before a definitive conclusion on possible health risks could be drawn. Although the toxicity of organotins has been extensively reviewed, the reproductive and developmental toxicity of organotins is not well understood. We summarized the data of the studies on reproductive and developmental toxicity of organotins in aquatic
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Hirose, A;
Takagi, A;
Nishimura, T;
Kanno, J;
Ema, M
[1]
- National Inst. of Health Sciences, Tokyo (Japan)
Citation Formats
Hirose, A, Takagi, A, Nishimura, T, Kanno, J, and Ema, M.
Review of reproductive and developmental toxicity induced by organotins in aquatic organisms and experimental animals.
Germany: N. p.,
2004.
Web.
Hirose, A, Takagi, A, Nishimura, T, Kanno, J, & Ema, M.
Review of reproductive and developmental toxicity induced by organotins in aquatic organisms and experimental animals.
Germany.
Hirose, A, Takagi, A, Nishimura, T, Kanno, J, and Ema, M.
2004.
"Review of reproductive and developmental toxicity induced by organotins in aquatic organisms and experimental animals."
Germany.
@misc{etde_20828294,
title = {Review of reproductive and developmental toxicity induced by organotins in aquatic organisms and experimental animals}
author = {Hirose, A, Takagi, A, Nishimura, T, Kanno, J, and Ema, M}
abstractNote = {Widespread use of organotins has caused increasing amounts to be released into the environment. The most important non-pesticidal route of entry of organotins into the environment is through leaching of organotin-stabilized PVC in water, and the use in antifouling agents, resulting in the introduction of organotin into the aquatic environment. Data are available regarding the detection of butyltins and phenyltins in aquatic marine organisms and marine products. Food chain bioamplification of butyltin in oysters, mud crabs, marine mussels, chinook salmons, dolphins, tunas, and sharks and of phenyltin in carps and horseshoe crabs has been reported. These findings indicate that organotins accumulate in the food chain and are bioconcentrated, and that humans can be exposed to organotins via seafood. The levels of organotin compounds in seafood are not considered to be sufficiently high to affect human health. However, Belfroid et al. (2000) noted that more research on residual TBT levels in seafood was needed before a definitive conclusion on possible health risks could be drawn. Although the toxicity of organotins has been extensively reviewed, the reproductive and developmental toxicity of organotins is not well understood. We summarized the data of the studies on reproductive and developmental toxicity of organotins in aquatic organisms and experimental animals.}
place = {Germany}
year = {2004}
month = {Sep}
}
title = {Review of reproductive and developmental toxicity induced by organotins in aquatic organisms and experimental animals}
author = {Hirose, A, Takagi, A, Nishimura, T, Kanno, J, and Ema, M}
abstractNote = {Widespread use of organotins has caused increasing amounts to be released into the environment. The most important non-pesticidal route of entry of organotins into the environment is through leaching of organotin-stabilized PVC in water, and the use in antifouling agents, resulting in the introduction of organotin into the aquatic environment. Data are available regarding the detection of butyltins and phenyltins in aquatic marine organisms and marine products. Food chain bioamplification of butyltin in oysters, mud crabs, marine mussels, chinook salmons, dolphins, tunas, and sharks and of phenyltin in carps and horseshoe crabs has been reported. These findings indicate that organotins accumulate in the food chain and are bioconcentrated, and that humans can be exposed to organotins via seafood. The levels of organotin compounds in seafood are not considered to be sufficiently high to affect human health. However, Belfroid et al. (2000) noted that more research on residual TBT levels in seafood was needed before a definitive conclusion on possible health risks could be drawn. Although the toxicity of organotins has been extensively reviewed, the reproductive and developmental toxicity of organotins is not well understood. We summarized the data of the studies on reproductive and developmental toxicity of organotins in aquatic organisms and experimental animals.}
place = {Germany}
year = {2004}
month = {Sep}
}