skip to main content
OSTI.GOV title logo U.S. Department of Energy
Office of Scientific and Technical Information

Title: Bipolar Membranes Inhibit Product Crossover in CO 2 Electrolysis Cells

 [1];  [1];  [1];  [2];  [2]; ORCiD logo [1]
  1. Department of Chemistry, The Pennsylvania State University, University Park PA 16802 USA
  2. Department of Chemical and Biomolecular Engineering, Vanderbilt University, Nashville TN 37235 USA
Publication Date:
Sponsoring Org.:
OSTI Identifier:
Grant/Contract Number:
Resource Type:
Journal Article: Publisher's Accepted Manuscript
Journal Name:
Advanced Sustainable Systems
Additional Journal Information:
Related Information: CHORUS Timestamp: 2018-03-02 03:54:15; Journal ID: ISSN 2366-7486
Wiley Blackwell (John Wiley & Sons)
Country of Publication:

Citation Formats

Li, Yuguang C., Yan, Zhifei, Hitt, Jeremy, Wycisk, Ryszard, Pintauro, Peter N., and Mallouk, Thomas E.. Bipolar Membranes Inhibit Product Crossover in CO 2 Electrolysis Cells. Germany: N. p., 2018. Web. doi:10.1002/adsu.201700187.
Li, Yuguang C., Yan, Zhifei, Hitt, Jeremy, Wycisk, Ryszard, Pintauro, Peter N., & Mallouk, Thomas E.. Bipolar Membranes Inhibit Product Crossover in CO 2 Electrolysis Cells. Germany. doi:10.1002/adsu.201700187.
Li, Yuguang C., Yan, Zhifei, Hitt, Jeremy, Wycisk, Ryszard, Pintauro, Peter N., and Mallouk, Thomas E.. 2018. "Bipolar Membranes Inhibit Product Crossover in CO 2 Electrolysis Cells". Germany. doi:10.1002/adsu.201700187.
title = {Bipolar Membranes Inhibit Product Crossover in CO 2 Electrolysis Cells},
author = {Li, Yuguang C. and Yan, Zhifei and Hitt, Jeremy and Wycisk, Ryszard and Pintauro, Peter N. and Mallouk, Thomas E.},
abstractNote = {},
doi = {10.1002/adsu.201700187},
journal = {Advanced Sustainable Systems},
number = ,
volume = ,
place = {Germany},
year = 2018,
month = 3

Journal Article:
Free Publicly Available Full Text
This content will become publicly available on March 2, 2019
Publisher's Accepted Manuscript

Save / Share:
  • C6 glioma cells respond to ..beta..-adrenergic agonists (isoproterenol) with a transient rise in intracellular cyclic adenosine monophosphate level. This ..beta..-responsiveness of C6 cells is inhibitied by the presence of a plasma membrane fraction, which has a five- to six-fold purification of membrane markers, showed a greater inhibition of ..beta..-responsiveness in C6 cells than any other subcellular fractions of B104 cells. The inhibitory effector(s) is apparently associated with integral membrane structure(s) since ionic extraction and treatment with chelating agents did not remove the effect from the particulate membrane fraction. The effector is probably proteinaceous in nature as judged by its susceptibilitymore » to inactivation by heat and protease treatment. The data indicate that neither adenylate cyclase nor phosphodiesterase enzyme is likely to be directly involved in mediating the ..beta..-nonresponsiveness of C6 cells.« less
  • New polymer electrolyte membranes for polymer-electrolyte-membrane (PEM) fuel cells (PEFCs) have been developed. Platinum nanocrystals (d = 1--2 nm) were highly dispersed in a Nafion 112 film (Pt-PEM, thickness 50 {micro}m) to catalyze the recombination of the crossover H{sub 2} with O{sub 2}, and the water generated was found to humidify the Pt-PEM directly. In order to clarify the self-humidifying properties in the Pt-PEM, the amount of water vapor produced by the recombination and the faradaic reaction was analyzed together with those of consumed H{sub 2} and O{sub 2} by monitoring humidity in the exhausting gases from PEFCs operated withmore » dry H{sub 2} consumption decreased to ca. 2/3 of that in normal membranes. All H{sub 2}O produced inside the Pt-PEM was found to be exhausted from the anode, resulting in the efficient humidification of the membrane on the anode side, which is dried by electro-osmotic drag. Thus, the resistance of the Pt-PEM was lowered to 0.04 {Omega} cm{sup 2}. It is also found that the Pt-PEM improved the cathode potential distinctively, which was ascribed to eliminate of the chemical reaction of crossover gases in the cathode catalyst layer, and eliminated any disturbance of O{sub 2} diffusion by H{sub 2}O vapor produced by the reaction. The operation of PEFCs with minimal or no humidification by using Pt-PEMs is essential in applications to power sources for electric vehicles or various electronic devices from the viewpoints of the simplification of control systems, cold starts, or response to abrupt load changes.« less
  • Interest has been growing in direct ethanol fuel cells (DEFCs) due to their non-toxicity, low cost and potential contribution to energy issues in third world countries. A reduction in fuel cross-over is of key importance to enhance the performance of DEFCs that operate at low temperatures (<100 °C). We report on the effect of the addition of phosphotungstic acid (PWA) in Nafion membrane on the ethanol-crossover for DEFC application. A set of PWANafion composite membranes (PWA 0, 5, 10, 15, 20 wt%) was prepared by solution casting and their microstructures, diffraction patterns and permeability were systematically characterized. The significant reductionmore » in ethanol-crossover was observed with increasing PWA concentration in PWA-Nafion membranes, which was mainly attributed to an improvement in crystallinity of the membrane. PWA provides additional nucleation sites during solidification leading to higher crystallinity, which is supported by the membrane permeability tests. These PWA-Nafion composites were implemented in proto-type DEFC devices as a membrane and the maximum power density achieved was 22% higher than that of commercial Nafion-117 device.« less
  • The relationship between binding of antipsychotic drugs and sigma psychotomimetic opiates to binding sites for the sigma agonist (+)-(/sup 3/H)SKF 10,047 (N-allylnormetazocine) and to dopamine D/sub 2/ sites was investigated. In guinea pig brain membranes, (+)-(/sup 3/H)SKF 10,047 bound to single class of sites with a K/sub d/ of 4 x 10/sup -8/ M and a B/sub max/ of 333 fmol/mg of protein. This binding was different from, kappa, or delta opiate receptor binding. It was inhibited by opiates that produce psychotomimetic activities but not by opiates that lack such activities. Some antipsychotic drugs inhibited (+)-(/sup 3/H)SKF 10,047 bindingmore » with high to moderate affinities in the following order of potency: haloperidol > perphenazine > fluphenazine > acetophenazine > trifluoperazine > molindone greater than or equal to pimozide greater than or equal to thioridazine greater than or equal to chlorpromazine greater than or equal to triflupromazine. However, there were other antipsychotic drugs such as spiperone and clozapine that showed low affinity for the (+)-(/sup 3/H)SKF 10,047 binding sites. Affinities of antipsychotic drugs for (+)-(/sup 3/H)SKF 10,047 binding sites did not correlate with those for (/sup 3/H)spiperone (dopamine D/sub 2/) sites. (/sup 3/H)-Haloperidol binding in whole brain membranes was also inhibited by the sigma opiates pentazocine, cyclazocine, and (+)-(/sup 3/H)SKF 10,047. In the striatum, about half of the saturable (/sup 3/H)haloperidol binding was to (/sup 3/H)spiperone (D/sub 2/) sites and the other half was to sites similar to (+)-(/sup 3/H)SKF 10,047 binding sites. 15 references, 4 figures, 1 table.« less
  • Alpha adrenergic agonists and antagonists as clonidine, guanfacine, yohimbine, phenylephrine and prazosin inhibited the ({sup 3}H)-QNB binding to rat brain cortex muscarinic acetylcholine receptor (mAChR, M-1 subtype), heart (M-2 subtype) and parotid gland homogenate (M-3 subtype) in a dose-dependent competitive fashion. Ki values were between 10{sup {minus}6} and 10{sup {minus}3} M. Hill coefficients were about 1. No correlation was found between mAChR inhibiting capacity of these drugs and their activity on alpha adrenergic receptors. In contrast, other transmitters, as dopamine, GABA, glutamic acid, histamine, serotonin, isoproterenol and platelet activating factor (PAF) did not affect the QNB binding.