Bruchas, Michael R. - Department of Anesthesiology, Washington University in St. Louis

Advanced Search

Bruchas, Michael R. - Department of Anesthesiology, Washington University in St. Louis

Michael R. Bruchas, Ph.D Assistant Professor
Research Report The dynorphin/kappa opioid system as a modulator of
CRF1-R Activation of the Dynorphin/Kappa Opioid System in the Mouse Basolateral Amygdala Mediates
Repeated swim stress induces kappa opioid-mediated activation of extracellular
Phosphorylation of the -Opioid Receptor at Tyrosine 166 (Tyr3.51) in the DRY Motif Reduces Agonist EfficacyS
Long-acting Opioid Antagonists Disrupt Receptor Signaling and Produce Noncompetitive Effects by Activating c-Jun
Kappa Opioid Receptor Activation of p38 MAPK Is GRK3-and Arrestin-dependent in Neurons and Astrocytes*
Tyrosine Phosphorylation of Kir3.1 in Spinal Cord Is Induced by Acute Inflammation, Chronic Neuropathic Pain, and
Behavioral/Systems/Cognitive Stress-Induced p38 Mitogen-Activated Protein Kinase
Ligand-directed c-Jun N-terminal kinase activation disrupts opioid receptor signaling
Kinase cascades and ligand-directed signaling at the kappa opioid receptor
Activation of the kappa opioid receptor in the dorsal raphe nucleus mediates the aversive effects of stress
Behavioral/Systems/Cognitive The Dysphoric Component of Stress Is Encoded by
Characterization of the 1-adrenoceptor subtype activating extracellular signal-regulated kinase in submandibular gland acinar cells
THE ABSENCE OF ENDOGENOUS -ENDORPHIN SELECTIVELY BLOCKS PHOSPHORYLATION AND DESENSITIZATION OF MU OPIOID
Development/Plasticity/Repair Sciatic Nerve Ligation-Induced Proliferation of Spinal Cord
Calmodulin-Stimulated Adenylyl Cyclase Gene Deletion Affects Morphine Responses
Submandibular Gland Acinar Cells Express Multiple 1-Adrenoceptor Subtypes
Selective p38a MAPK Deletion in Serotonergic Neurons Produces Stress Resilience