Bielas, Jason H. - Bielas, Jason H. - Public Health Sciences, Fred Hutchinson Cancer Research Center, University of Washington at Seattle

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Bielas, Jason H. - Bielas, Jason H. - Public Health Sciences, Fred Hutchinson Cancer Research Center, University of Washington at Seattle

dna repair 5 ( 2 0 0 6 ) 294302 available at www.sciencedirect.com
Review Article Generation, Function, and Prognostic Utility of Somatic
Brief Report Molecularly Evolved Thymidylate Synthase Inhibits
This article appeared in a journal published by Elsevier. The attached copy is furnished to the author for internal non-commercial research
The most fundamental problem with the mutator phenotype hypothesis is that it simply does not take adequate account of the
Canada and the Tula Foundation. P.J.K. is a Fellow of the Canadian Institute for Advanced Research and a Michael Smith Foundation for
Human cancers express a mutator phenotype Jason H. Bielas, Keith R. Loeb*, Brian P. Rubin, Lawrence D. True, and Lawrence A. Loeb
Perspective Unifying Concept of DNA Repair
Perspective MutatorPhenotypeinCancer: Timing and Perspectives
DNA Repair 3 (2004) 711717 Quiescent murine cells lack global genomic repair but are
Elevated mutagenesis and decreased DNA repair at a transgene are associated with proliferation but not
Mutation Research 518 (2002) 107112 A more efficient Big Blue protocol improves transgene rescue
Proliferation is necessary for both repair and mutation in transgenic mouse cells
DNA deletions and clonal mutations drive premature aging
Cancers Exhibit a Mutator Phenotype: Clinical Implications Lawrence A. Loeb,
The cII Locus in the MutaTM Mouse System
Mitochondrial point mutations do not limit the natural lifespan of mice
9 FEBRUARY 2007 VOL 315 SCIENCE www.sciencemag.org762 LETTERS I BOOKS I POLICY FORUM I EDUCATION FORUM I PERSPECTIVES
Non-transcribed strand repair revealed in quiescent cells Jason H. Bielas*
Correction: Genetic Instability Is Not a Requirement for Tumor Development