Tabas, Ira - Department of Physiology and Cellular Biophysics, Columbia University

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Tabas, Ira - Department of Physiology and Cellular Biophysics, Columbia University

JCB: Article The Rockefeller University Press $30.00
Cell Metabolism Atherogenic Lipids and Lipoproteins Trigger
Role of cholesterol and lipid organization in Frederick R. Maxfield1
Cell Metabolism Short Article
Sitosterol-containing Lipoproteins Trigger Free Sterol-induced Caspase-independent Death in ACAT-competent Macrophages*
Cholesterol-induced Apoptotic Macrophages Elicit an Inflammatory Response in Phagocytes, Which Is Partially
A R T I C L E S NATURE CELL BIOLOGY VOLUME 5 | NUMBER 9 | SEPTEMBER 2003 781
This article is available online at http://www.jlr.org Journal of Lipid Research Volume 52, 2011 1033 Copyright 2011 by the American Society for Biochemistry and Molecular Biology, Inc.
The cells of most organs and tissues satisfy their require-ments for membrane cholesterol via endogenous cho-
Imagine if an excess amount of a critical, life-sustain-ing molecule like ATP were, by a perverse series of
Free Cholesterol-loaded Macrophages Are an Abundant Source of Tumor Necrosis Factor-and Interleukin-6
THEJOURNALOFCELLBIOLOGY The Rockefeller University Press $8.00
macrophages Combinatorial pattern recognition receptor signaling alters the balance of life and death in
Mechanisms and consequences of efferocytosis in advanced atherosclerosis
Cell Metabolism IRE1b Inhibits Chylomicron Production
Integrating the mechanisms of apoptosis induced by endoplasmic reticulum stress
The successful resolution of inflammatory disease processes, often known as `catabasis', requires a distinct
L E T T E R S Adaptive suppression of the ATF4CHOP branch of the
FORUM REVIEW ARTICLE Macrophage Apoptosis in Atherosclerosis
Research article TheJournalofClinicalInvestigation http://www.jci.org Volume119 Number10 October2009 2925
The Rockefeller University Press $30.00 J. Cell Biol. Vol. 186 No. 6 783792
Extracellular Nampt Promotes Macrophage Survival via a Nonenzymatic Interleukin-6/STAT3 Signaling Mechanism*
Pivotal Advance: Macrophages become resistant to cholesterol-induced death after phagocytosis of
2006LANDESBIOSCIENCE.DONOTDISTRIBUTE. [Autophagy 3:1, 38-41, January/February 2007]; 2007 Landes Bioscience
Consequences and Therapeutic Implications of Macrophage Apoptosis in Atherosclerosis
NiemannPick C heterozygosity confers resistance to lesional necrosis and macrophage apoptosis in
Enrichment of Endoplasmic Reticulum with Cholesterol Inhibits Sarcoplasmic-Endoplasmic Reticulum Calcium ATPase-2b Activity
News and Commentary Apoptosis and plaque destabilization in
Sphingolipids and Atherosclerosis A Mechanistic Connection? A Therapeutic Opportunity?
The Cytoplasmic Domain of the Low Density Lipoprotein (LDL) Receptor-related Protein, but Not That of the LDL Receptor,
Leading Edge Macrophages in the Pathogenesis
Traffic 2010; 11: 601615 2010 John Wiley & Sons A/S doi:10.1111/j.1600-0854.2010.01046.x
Suppression of Macrophage Eicosanoid Synthesis by Atherogenic Lipoproteins Is Profoundly Affected by Cholesterol-Fatty Acyl
ABCA1-mediated Cholesterol Efflux Is Defective in Free Cholesterol-loaded Macrophages
Research article 886 TheJournalofClinicalInvestigation http://www.jci.org Volume 119 Number 4 April 2009
Acid Sphingomyelinase-deficient Macrophages Have Defective Cholesterol Trafficking and Efflux*
SHEDDING OF THE MER TYROSINE KINASE RECEPTOR IS MEDIATED BY ADAM17 THROUGH A PATHWAY INVOLVING REACTIVE OXYGEN SPECIES,
B e t w e e n B e d s i d e an d B e n c h atherosclerosis than those reconstituted with
Cell Metabolism Insulin Resistance, Hyperglycemia,