- Dmitri LODYGIN and Heiko HERMEKING www.cell-research.com | Cell Research, 15(4):237-246, Apr 2005
- Inducible microRNA expression by an all-in-one episomal vector system
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- Signalling pathways that normally prevent deregulated proliferation or contribute to sensitivity to apoptotic
- Serial analysis of gene expression and cancer Heiko Hermeking, PhD
- The G2/M Regulator 14-3-3 Prevents Apoptosis through Sequestration of Bax*
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- High frequency of hypermethylation at the 14-3-3 locus leads to gene silencing in breast cancer
- Identification of CDK4 as a target of c-MYC Heiko Hermeking*
- Molecular Cell, Vol. 1, 311, December, 1997, Copyright 1997 by Cell Press 14-3-3r Is a p53-Regulated Inhibitor
- Seminars in Cancer Biology 16 (2006) 161 14-3-3 proteins and cancer biology
- Analysis of 14-3-3r expression in hyperproliferative skin diseases reveals selective loss associated with CpG-methylation in basal cell carcinoma
- [CANCER RESEARCH 62, 62556262, November 1, 2002] Characterization of Epithelial Senescence by Serial Analysis of Gene Expression
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- Cancer Cell 414 CancerCell12,November20072007ElsevierInc.
- CBPcmediated transcription but activated transcription mediated by the prototypical tran-
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- Induction of Cullin 7 by DNA damage attenuates p53 function
- 1999 Macmillan Magazines Ltd letters to nature
- 2007LANDESBIOSCIENCE.DONOTDISTRIBUTE. Digital Karyotyping Reveals Frequent Inactivation of the Dystrophin/
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- Current Cancer Drug Targets, 2003, 3, 163-175 163 1568-0096/03 $41.00+.00 2003 Bentham Science Publishers Ltd.
- Targeted Proteomic Analysis of 14-3-3 , a p53 Effector Commonly Silenced in Cancer*S
- 2007LANDESBIOSCIENCE.DONOTDISTRIBUTE. Large-Scale Identification of c-MYC-Associated Proteins
- Functional Epigenomics Identifies Genes Frequently Silenced in Prostate Cancer
