
- Immunity, Vol. 13, 13, July, 2000, Copyright 2000 by Cell Press Xid-like Phenotypes: A B Cell Minireview
- FOXO Transcription Factors Cooperate with EF1 to Activate Growth Suppressive Genes in B Lymphocytes1
- Phosphoinositide 3-kinase signaling is essential for ABL oncogenemediated transformation of B-lineage cells
- nature genetics volume 26 november 2000 379 Hypoglycaemia, liver necrosis and perinatal death in mice
- Impaired B Cell Development and Proliferation in Absence of
- Research article 3038 TheJournalofClinicalInvestigation http://www.jci.org Volume 118 Number 9 September 2008
- Proliferation and Survival of Activated B Cells Requires Sustained Antigen Receptor Engagement and Phosphoinositide
- T Cell Ig and Mucin Domain-1-Mediated T Cell Activation Requires Recruitment and Activation of Phosphoinositide
- IMMUNOBIOLOGY T-cell function is partially maintained in the absence of class IA
- International Immunology, Vol. 20, No. 5, pp. 671681 doi:10.1093/intimm/dxn024
- Inflammation 1109 Lymphocyte cell motility: the twisting, turning
- C H A P T E R E I G H T Measuring Phosphorylated Akt
- This article was downloaded by:[CDL Journals Account] On: 29 August 2007
- 3rd Focused Meeting on PI3K Signalling and Disease 177 The role of class I phosphoinositide 3-kinase in
- Distinct signaling mechanisms activate the target of rapamycin in response to different B-cell stimuli
- Class IA Phosphoinositide 3-Kinase Modulates Basal Lymphocyte Motility in the Lymph Node1
- KLF4 suppresses transformation of pre-B cells by ABL oncogenes Michael G. Kharas,1 Isharat Yusuf,1 Vanessa M. Scarfone,1 Vincent W. Yang,2 Julia A. Segre,3 Claudia S. Huettner,4
- phosphoinositide-3-kinase Sjgren's syndrome-like disease in mice with T cells lacking class 1A
- IMMUNOBIOLOGY Brief report
- Altered splenic B cell subset development in mice lacking phosphoinositide 3-kinase p85a
- Phosphoinositide 3-kinase and its targets in B-cell and T-cell signaling
- Seminars in Cell & Developmental Biology 15 (2004) 183197 PI3K signaling controls cell fate at many points
- Annu. Rev. Immunol. 2004. 22:56398 doi: 10.1146/annurev.immunol.22.012703.104721
- PI-3KinaseinSignallingandDiseaseBiochemicalSocietyFocusedMeeting PI-3 Kinase in Signalling and Disease
- Phosphoinositide 3-kinase and Bruton's tyrosine kinase regulate overlapping sets of genes in
- doi:10.1006/smim.2001.0337, available online at http://www.idealibrary.com on seminars in IMMUNOLOGY, Vol. 14, 2002: pp. 718
- Impaired Kit-but Not Fc RI-initiated Mast Cell Activation in the Absence of Phosphoinositide 3-Kinase p85 Gene Products*
- ABL Oncogenes and Phosphoinositide 3-Kinase: Mechanism of Activation and Downstream Effectors
- Introduction Insulin resistance is a major pathophysiologic feature
- Enhanced T Cell Proliferation in Mice Lacking the p85 Subunit of Phosphoinositide 3-Kinase1
- Regulation of quiescence in lymphocytes