- Regulation of the Caenorhabditis elegans oxidative stress defense protein SKN-1
- RNAi Screening Implicates a SKN-1Dependent Transcriptional Response in Stress Resistance and
- INTRODUCTION Germ cells are both pluripotent and immortal, because they
- THEJOURNALOFCELLBIOLOGY JCB: ARTICLE
- Molecular Cell, Vol. 14, 553557, June 4, 2004, Copyright 2004 by Cell Press Letter to the Editor
- The Caenorhabditis elegans mRNA 5 -Capping Enzyme IN VITRO AND IN VIVO CHARACTERIZATION*
- Similar but distinct eects of the tristetraprolin/TIS11 immediate-early proteins on cell survival
- PERSPECTIVE Getting the right dose of repression
- SKN-1 domain folding and basic region monomer stabilization upon DNA binding
- Requirement for P Granules and Meiosis for Accumulation of the Germline RNA Helicase CGH-1
- Edited by Judith Kimble and Susan Strome. Last revised June 3, 2008. Published September 4, 2008. This chapter should be cited as: Gartner et al. Germline Survival and Apoptosis (September 4, 2008), WormBook, ed. The C. elegans Research Community, WormBoo
- Transcriptional repression by the Caenorhabditis elegans
- A Conserved Transcription Motif Suggesting Functional Parallels between Caenorhabditis elegans SKN-1 and Cap`n'Collar-related
- MOLECULAR AND CELLULAR BIOLOGY, 0270-7306/99/$04.00 0
- MK2-induced tristetraprolin:14-3-3 complexes prevent stress granule association and ARE-mRNA
- A Broad but Restricted Requirement for TAF-5 (Human TAFII100) for Embryonic Transcription in Caenorhabditis elegans*
- Transcription Elongation: TLKing to T. Keith Blackwell and Amy K. Walker
- Cell Metabolism Life Is Short, if Sweet
- Condition-adapted stress and longevity gene regulation by Caenorhabditis elegans SKN-1/Nrf
- A redox-sensitive peroxiredoxin that is important for longevity has tissue-and stress-specific roles in
- 18 Cell 135, October 3, 2008 2008 Elsevier Inc. Bershadsky, A., Chausovsky, A., Becker, E., Lyu-
- Direct Inhibition of the Longevity-Promoting Factor SKN-1
- Transcription reactivation steps stimulated by oocyte maturation in C. elegans
- A Conserved MST-FOXO Signaling Pathway Mediates Oxidative-Stress
- Edited by Thomas Blumenthal. Last revised July 14, 2006. Published September 5, 2006. This chapter should be cited as: Blackwell, T. K. and Walker, A. K. Transcription mechanisms (September 5, 2006), WormBook, ed. The C. elegans Research Community, WormBo
- RESEARCH COMMUNICATION The C. elegans p38 MAPK
- Introduction Germ cells are a highly specialized lineage that is responsible
- An Extensive Requirement for Transcription Factor IID-specific TAF-1 in Caenorhabditis elegans Embryonic Transcription*
- The Caenorhabditis elegans Ortholog of TRAP240, CeTRAP240/let-19, Selectively Modulates Gene Expression and Is Essential
- Germ Cells: Finding Programs of Mass T. Keith Blackwell
- Molecular Cell of thymocyte development, the E47 gene products form to dimerize and severely affect the ability to induce poly-
- A model of repression: CTD analogs and PIE-1 inhibit transcriptional
- Broad Requirement for the Mediator Subunit RGR-1
- CDK-9/cyclin T (P-TEFb) is required in two postinitiation pathways
- Multiple tristetraprolin sequence domains required to induce apoptosis and modulate responses to TNFa through distinct pathways
- MOLECULAR AND CELLULAR BIOLOGY, 0270-7306/00/$04.00 0
- Gene 228 (1999) 133145 Identification of four CCCH zinc finger proteins in Xenopus,
- Cytoplasmic Localization of Tristetraprolin Involves 14-3-3-dependent and -independent Mechanisms*
- Specific SKN-1/Nrf Stress Responses to Perturbations in Translation Elongation and Proteasome Activity
