- Molecular Cell, Vol. 10, 15031509, December, 2002, Copyright 2002 by Cell Press Drosophila MUS312 Interacts with the
- Perspective Mitotic Recombination: Why? When? How? Where?
- Prospects & Overviews Meiotic versus mitotic recombination
- Cytogenet Genome Res 107:172179 (2004) DOI: 10.1159/000080595
- Z .Mutation Research 459 2000 219228 www.elsevier.comrlocaterdnarepair
- 257 (1995); M. S. Swanson and R. R. Isberg, Infect. Immun. 64, 2585 (1995).
- Cell, Vol. 83, 157-160, October 20, 1995, Copyright 0 1995 by Cell Press The Bond between Sisters
- Copright 0 1995 by the Genetics Society of America The Drosophila Meiotic Recombination Gene mei-7 Encodes a Homologue
- Abstract. This paper reports on a new role for mei-41 in cell cycle control during meiosis. This function is re-
- The Rockefeller University Press, 0021-9525/2000/07/F31/6 $5.00 The Journal of Cell Biology, Volume 150, Number 2, July 24, 2000 F31F36
- Molecular Cell Short Article
- mus304 encodes a novel DNA damage checkpoint protein required during
- Interstrand crosslink repair: can XPF-ERCC1 be let off the hook?
- REC, Drosophila MCM8, Drives Formation of Meiotic Crossovers
- DNA Repair 8 (2009) 803812 Contents lists available at ScienceDirect
- Copyright 2007 by the Genetics Society of America DOI: 10.1534/genetics.106.070060
- Copyright 2007 by the Genetics Society of America DOI: 10.1534/genetics.107.073635
- Formation of deletions during double-strand break repair in Drosophila DmBlm mutants occurs after
- 2002 Macmillan Magazines Ltd Drosophila melanogaster has been an important model
- Copyright 1999 by the Genetics Society of America Identification of Novel Drosophila Meiotic Genes
- Copyright 1998 by the Genetics Society of America Damage Control: The Pleiotropy of DNA Repair Genes in Drosophila melanogaster
- Copyright 0 1995 by the Genetics Society of America Genetic Characterization and Cloningof Mothers against dpp, a Gene
- Transcription Initiation From Within P Elements Generates Hypomorphic Mutations in Drosophila melanogaster
- Three Structure-Selective Endonucleases Are Essential in the Absence of BLM Helicase in Drosophila
