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53BP1 and p53 synergize to suppress genomic instability and lymphomagenesis
 

Summary: 53BP1 and p53 synergize to suppress genomic
instability and lymphomagenesis
Julio C. Morales*, Sonia Franco
, Michael M. Murphy
, Craig H. Bassing
, Kevin D. Mills§
, Melissa M. Adams*,
Nicole C. Walsh¶
, John P. Manis¶
, George Z. Rassidakis , Frederick W. Alt
**
, and Phillip B. Carpenter*,
**
*Department of Biochemistry and Molecular Biology, University of Texas Health Sciences Center, Houston, TX 77030; Center for Blood Research, Children's
Hospital, Howard Hughes Medical Institute, Harvard Medical School, Boston, MA 02115; ¶Joint Program in Transfusion Medicine, Department of Laboratory
Medicine, Children's Hospital, and Department of Pathology, Harvard Medical School, Boston, MA 02115; and Department of Hematopathology,
University of Texas M. D. Anderson Cancer Center, Houston, TX 77030
Contributed by Frederick W. Alt, December 28, 2005
p53-binding protein 1 (53BP1) participates in the cellular response
to DNA double-stranded breaks where it associates with various

  

Source: Alt,, Frederick - Immune Disease Institute, Harvard University

 

Collections: Biology and Medicine